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Interestingly, TNBC had the highest expression of GLUT1 when contrasted with other subtypes, suggesting the highly active metabolic status in TNBC (18)

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Specific transport proteins, such as organic anion transporting polypeptides ( OTAP1B1 and OATP1B3 ), actively facilitate the transfer of bilirubin from the sinusoidal blood into hepatocytes [24]

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But its GI side effect profile is also the most aggressive, with nausea rates reaching 40% to 50% and vomiting rates significantly higher than cagrilintide monotherapy